Portional to the number of subjects who have the indicated event rateA variety of point mutations in the regulatory elements of the g-globin genes (HBG1, HBG2), especially, give rise to non-gene-deletion forms HPFH and can be co-inherited with the bS allele,12 and these define the classical sickle bglobin haplotypes. The laboratory identification of these haplotypes (e.g. Benin, Senegal, Central A
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